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  Rituximab (a humanized anti-CD20 monoclonal antibody) is a useful remedy for Epstein-Barr virus-associated submit transplant lymphoproliferative illness (EBV-PTLD).
  Nevertheless, hypogammaglobulinemia is a complication related to this treatment.1,2 Little is thought in regards to the duration and severity of rituximab-induced low serum immunoglobulin (Ig) standing.
  EBV-PTLD. Considered one of three patients reported
  by Verschuuren et al1 died of invasive aspergillosis
  resulting from hypogammaglobulinemia after 7 months of rituximab remedy.
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  Verschuuren EA, Stevens SJ, van Imhoff GW et al.
  Remedy of posttransplant lymphoproliferative illness with
  rituximab: the remission, the relapse, and the complication. Quartier P,
  Brethon B, Philippet P et al. Therapy of childhood autoimmune
  haemolytic anaemia with rituximab. Castagnola E, Dallorso S, Faraci M et al.
  Lengthy-lasting hypogammaglobulinemia following rituximab administration for Epstein-Barr virus-associated put up-transplant lymphoproliferative disease preemptive therapy.
  Imashuku S, Naya M, Tamura S et al. Effectiveness of
  rituximab for chemotherapy-resistant multiple tumoral B-LPD in a haematopoietic stem cell recipient.
  Faye A, Quartier P, Reguerre Y et al. Chimaeric anti-CD20 monoclonal
  antibody (rituximab) in publish-transplant B-lymphoproliferative disorder following stem cell
  transplantation in kids. Esser JWJ, Niesters HGM, van der Holt
  B et al. Prevention of Epstein-Barr virus-lymphoproliferative disease by molecular monitoring and preemptive rituximab in high-risk patients after allogeneic stem cell transplantation. Kuehnle
  I, Huls MH, Liu Z et al. CD20 monoclonal antibody (rituximab) for therapy of
  Epstein-Barr virus lymphoma after hemopoietic
  stem cell transplantation. Imashuku, S.,
  Teramura, T., Morimoto, A. et al. Extended hypogammaglobulinemia following rituximab therapy for submit transplant Epstein-Barr
  virus-related lymphoproliferative disease.
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